Sanz Bueno, Jimena Cullen, Daniella Zarco Olivo, Carlos Vanaclocha Sebastián, Francisco
Paraneoplastic autoimmune multiorgan syndrome (paraneoplastic pemphigus) with unusual manifestations and without detectable autoantibodies. [caso clínico] - Indian journal of dermatology, venereology and leprology 2014 - 80(4):328-30.
Formato Vancouver:
Sanz Bueno J, Cullen D, Zarco C, Vanaclocha F. Paraneoplastic autoimmune multiorgan syndrome (paraneoplastic pemphigus) with unusual manifestations and without detectable autoantibodies. Indian J Dermatol Venereol Leprol. 2014 Jul-Aug;80(4):328-30.
PMID: 25035358
Contiene 9 referencias
We describe a patient with paraneoplastic autoimmune multiorgan syndrome (PAMS) secondary to a lymphoblastic T- cell lymphoma who presented with a lichenoid dermatitis and vitiligo, later developing bronchiolitis obliterans and autoimmune hepatitis. Notably, he had no detectable autoantibodies. The development of vitiligo and autoimmune hepatic involvement probably indicate a role for cytotoxic T- cell lymphocytes in the pathogenesis of this syndrome.
Paraneoplastic autoimmune multiorgan syndrome (paraneoplastic pemphigus) with unusual manifestations and without detectable autoantibodies. [caso clínico] - Indian journal of dermatology, venereology and leprology 2014 - 80(4):328-30.
Formato Vancouver:
Sanz Bueno J, Cullen D, Zarco C, Vanaclocha F. Paraneoplastic autoimmune multiorgan syndrome (paraneoplastic pemphigus) with unusual manifestations and without detectable autoantibodies. Indian J Dermatol Venereol Leprol. 2014 Jul-Aug;80(4):328-30.
PMID: 25035358
Contiene 9 referencias
We describe a patient with paraneoplastic autoimmune multiorgan syndrome (PAMS) secondary to a lymphoblastic T- cell lymphoma who presented with a lichenoid dermatitis and vitiligo, later developing bronchiolitis obliterans and autoimmune hepatitis. Notably, he had no detectable autoantibodies. The development of vitiligo and autoimmune hepatic involvement probably indicate a role for cytotoxic T- cell lymphocytes in the pathogenesis of this syndrome.
