Ruiz Hurtado, Gema
Dissecting the genetic predisposition to albuminuria and endothelial dysfunction in a genetic rat model. [artículo] - Journal of hypertension, 2013 - 31(11):2203-12; discussion 2212.
Formato Vancouver:
Steireif C, García-Prieto CF, Ruiz-Hurtado G, Pulido-Olmo H, Aranguez I, Gil-Ortega M et al. Dissecting the genetic predisposition to albuminuria and endothelial dysfunction
in a genetic rat model. J Hypertens. 2013 Nov;31(11):2203-12; discussion 2212.
PMID: 23868088
Contiene 40 referencias
OBJECTIVE: The Munich Wistar Frömter (MWF) rat develops progressive spontaneous albuminuria largely attributable to quantitative trait loci on rat chromosome (RNO)6 and RNO8, respectively. We tested the hypothesis whether quantitative trait loci on these chromosomes are linked to both albuminuria and endothelial dysfunction.
Dissecting the genetic predisposition to albuminuria and endothelial dysfunction in a genetic rat model. [artículo] - Journal of hypertension, 2013 - 31(11):2203-12; discussion 2212.
Formato Vancouver:
Steireif C, García-Prieto CF, Ruiz-Hurtado G, Pulido-Olmo H, Aranguez I, Gil-Ortega M et al. Dissecting the genetic predisposition to albuminuria and endothelial dysfunction
in a genetic rat model. J Hypertens. 2013 Nov;31(11):2203-12; discussion 2212.
PMID: 23868088
Contiene 40 referencias
OBJECTIVE: The Munich Wistar Frömter (MWF) rat develops progressive spontaneous albuminuria largely attributable to quantitative trait loci on rat chromosome (RNO)6 and RNO8, respectively. We tested the hypothesis whether quantitative trait loci on these chromosomes are linked to both albuminuria and endothelial dysfunction.
