Dissecting the genetic predisposition to albuminuria and endothelial dysfunction in a genetic rat model. [artículo]
Por: Ruiz Hurtado, Gema [Instituto de Investigación i+12]
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Colaborador(es): Instituto de Investigación imas12.
Editor: Journal of hypertension, 2013Descripción: 31(11):2203-12; discussion 2212.Recursos en línea: Solicitar documento Resumen: OBJECTIVE: The Munich Wistar Frömter (MWF) rat develops progressive spontaneous albuminuria largely attributable to quantitative trait loci on rat chromosome (RNO)6 and RNO8, respectively. We tested the hypothesis whether quantitative trait loci on these chromosomes are linked to both albuminuria and endothelial dysfunction.
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Artículo
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Formato Vancouver:
Steireif C, García-Prieto CF, Ruiz-Hurtado G, Pulido-Olmo H, Aranguez I, Gil-Ortega M et al. Dissecting the genetic predisposition to albuminuria and endothelial dysfunction
in a genetic rat model. J Hypertens. 2013 Nov;31(11):2203-12; discussion 2212.
PMID: 23868088
Contiene 40 referencias
OBJECTIVE: The Munich Wistar Frömter (MWF) rat develops progressive spontaneous albuminuria largely attributable to quantitative trait loci on rat chromosome (RNO)6 and RNO8, respectively. We tested the hypothesis whether quantitative trait loci on these chromosomes are linked to both albuminuria and endothelial dysfunction.
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