000			02844na a2200313 4500
999			_c1590 _d1590
003			PC1590
005			20210625062756.0
008			130622s2013 xxx||||| |||| 00| 0 eng d
040			_cH12O
041			_aeng
100			_aFiuza Luces, Carmen _92430 _eInstituto de Investigación i+12
100			_aLucía, Alejandro _92429 _eInstituto de Investigación i+12
100			_aMorán Jiménez, María Josefa _91013 _eInstituto de Investigación i+12
245	0	0	_aExercise training can induce cardiac autophagy at end-stage chronic conditions: Insights from a graft-versus-host-disease mouse model. _h[artículo]
260			_bBrain, behavior, and immunity, _c2014
300			_a39:56-60.
500			_aFormato Vancouver: Fiuza-Luces C, Delmiro A, Soares-Miranda L, González-Murillo Á, Martínez-Palacios J, Ramírez M et al. Exercise training can induce cardiac autophagy at end-stage chronic conditions: insights from a graft-versus-host-disease mouse model. Brain Behav Immun. 2014 Jul;39:56-60.
501			_aPMID: 24239952
504			_aContiene 35 referencias
520			_aINTRODUCTION: Chronic graft-versus-host disease (cGVHD) is a frequent cause of morbimortality after allogeneic hematopoietic stem cell transplantation (allo-HSCT), and severely compromises patients' physical capacity. Despite the aggressive nature of the disease, aerobic exercise training can positively impact survival as well as clinical and functional parameters. We analyzed potential mechanisms underlying the recently reported cardiac function improvement in an exercise-trained cGVHD murine model receiving lethal total body irradiation and immunosuppressant treatment (Fiuza-Luces et al., 2013. Med Sci Sports Exerc 45, 1703-1711). We hypothesized that a cellular quality-control mechanism that is receiving growing attention in biomedicine, autophagy, was involved in such improvement.
710			_9625 _aInstituto de Investigación imas12
856			_uhttp://pc-h12o-es.m-hdoct.a17.csinet.es/pdf/pc/1/pc1590.pdf _ySolicitar documento
942			_n0 _2ddc _cART