Inhibition of focal adhesion kinase prevents experimental lung fibrosis and myofibroblast formation. [artículo]
Por: Carreira Delgado, Patricia Esmeralda [Reumatología]
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Colaborador(es): Servicio de Reumatología.
Editor: Arthritis and Rheumatism, 2012Descripción: 64(5):1653-64.Recursos en línea: Acceso libre Resumen: OBJECTIVE: Enhanced adhesive signaling, including activation of focal adhesion kinase (FAK), is a hallmark of fibroblasts from lung fibrosis patients, and FAK has therefore been hypothesized to be a key mediator of this disease. This study was undertaken to characterize the contribution of FAK to the development of pulmonary fibrosis both in vivo and in vitro.
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Artículo
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Formato Vancouver:
Lagares D, Busnadiego O, García-Fernández RA, Kapoor M, Liu S, Carter DE, et al. Inhibition of focal adhesion kinase prevents experimental lung fibrosis and myofibroblast formation. Arthritis Rheum. 2012
May;64(5):1653-64.
PMID: 22492165
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OBJECTIVE: Enhanced adhesive signaling, including activation of focal adhesion kinase (FAK), is a hallmark of fibroblasts from lung fibrosis patients, and FAK has therefore been hypothesized to be a key mediator of this disease. This study was undertaken to characterize the contribution of FAK to the development of pulmonary fibrosis both in vivo and in vitro.
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