Dos glomerulonefritis en un mismo paciente con infección por virus de la hepatitis C. [artículo]
Por: Bengoa, Ignacio [Nefrología]
| Gutiérrez Martínez, Eduardo [Nefrología] | Hernández Martínez, Eduardo [Nefrología] | Martínez González, Miguel Ángel [Anatomía Patológica] | Molina Gómez, María [Nefrología] | Praga Terente, Manuel [Nefrología] | Sánchez Gómez, Julián [Anatomía Patológica] | Sevillano Prieto, Ángel Manuel [Nefrología].
Colaborador(es): Servicio de Anatomía Patológica | Servicio de Nefrología.
Editor: Nefrología : publicación oficial de la Sociedad Española Nefrologia, 2013Descripción: 33(4):593-600.Recursos en línea: Solicitar documento Resumen: Membranoproliferative glomerulonephritis (GN) may be pathogenically associated with infection due to the hepatitis C virus (HCV) as many clinical cases have shown. The potential relationship between HCV and IgA GN, by contrast, has been suggested only in isolated cases. IgA nephropathy recurs in up to 50% of cases after renal transplantation, but it rarely appears as a de novo GN. We report the case of a patient with chronic infection due to HCV and liver disease, who developed two different glomerular diseases during its evolution: membranoproliferative GN in their native kidneys, a process that caused end stage renal disease disease and de novo IgA GN in the transplanted kidney. The potential pathogenic relationship between both glomerular processes with infection due to HCV is discussed.
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Formato Vancouver:
Molina M, Sánchez J, Sevillano ÁM, Bengoa I, Gutiérrez E, Martínez MA et al. Dos glomerulonefritis en un mismo paciente con infección por virus de la hepatitis C. Nefrologia. 2013;33(4):593-600.
PMID: 23897193
Contiene 30 referencias
Membranoproliferative glomerulonephritis (GN) may be pathogenically associated with infection due to the hepatitis C virus (HCV) as many clinical cases have shown. The potential relationship between HCV and IgA GN, by contrast, has been suggested only in isolated cases. IgA nephropathy recurs in up to 50% of cases after renal transplantation, but it rarely appears as a de novo GN. We report the case of a patient with chronic infection due to HCV and liver disease, who developed two different glomerular diseases during its evolution: membranoproliferative GN in their native kidneys, a process that caused end stage renal disease disease and de novo IgA GN in the transplanted kidney. The potential pathogenic relationship between both glomerular processes with infection due to HCV is discussed.
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