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Intracranial internal carotid artery angioplasthy and stenting in giant cell arteritis. [caso clínico]

Por: Méndez Guerrero, Antonio [Neurología ] | Sierra Hidalgo, Fernando [Instituto de Investigación i+12] | Calleja Castaño, Patricia [Neurología] | Navia Alvárez, Pedro [Radiodagnóstico] | Campollo Velarde, Jorge [Radiodiagnóstico] | Díaz Guzmán, Jaime [Neurología].
Colaborador(es): Servicio de Neurología-Neurofisiología | Instituto de Investigación imas12 | Servicio de Radiodiagnóstico.
Tipo de material: materialTypeLabelArtículoEditor: Journal of neuroimaging : official journal of the American Society of Neuroimaging, 2015Descripción: 25(2):307-309.Recursos en línea: Solicitar documento Resumen: We report the case of a 59-year-old woman who presented with several episodes of transient ischemic attack (TIA) caused by pathologically confirmed giant cell arteritis. She continued suffering from TIAs during admission despite immunosuppressant and antithrombotic therapy. Sudden neurological deterioration with paraparesis and cognitive impairment developed. A brain magnetic resonance (MR) imaging showed bilateral watershed ischemic lesions. MR angiography demonstrated severe stenosis of both intracranial internal carotid arteries (ICAs). Angioplasty and stenting on the left ICA were performed, with evident clinical improvement occurring within 24 hours. Endovascular therapy may be an alternative option to treat severe GCA with symptomatic intracranial large vessel disease not responsive to intensive conventional medical treatment.
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Caso clínico Caso clínico PC17115 (Navegar estantería) Disponible

Formato Vancouver:
Guerrero AM, Sierra Hidalgo F, Calleja P, Navia P, Campollo J, Díaz Guzmán J. Intracranial internal carotid artery angioplasthy and stenting in giant cell arteritis. J Neuroimaging. 2015 Mar-Apr;25(2):307-309.

PMID: 24707958

Contiene 10 referencias

We report the case of a 59-year-old woman who presented with several episodes of transient ischemic attack (TIA) caused by pathologically confirmed giant cell arteritis. She continued suffering from TIAs during admission despite immunosuppressant and antithrombotic therapy. Sudden neurological deterioration with paraparesis and cognitive impairment developed. A brain magnetic resonance (MR) imaging showed bilateral watershed ischemic lesions. MR angiography demonstrated severe stenosis of both intracranial internal carotid arteries (ICAs). Angioplasty and stenting on the left ICA were performed, with evident clinical improvement occurring within 24 hours. Endovascular therapy may be an alternative option to treat severe GCA with symptomatic intracranial large vessel disease not responsive to intensive conventional medical treatment.

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