Stress-induced neuroinflammation: role of the Toll-like receptor-4 pathway. [artículo]
Por: Caso, Javier Rubén [Instituto de Investigación i+12]
| Gárate, Iciar [Instituto de Investigación i+12] | García Bueno, Borja [Instituto de Investigación i+12] | Leza, Juan Carlos [Instituto de Investigación i+12] | Muñoz Madrigal, José Luis [Instituto de Investigación i+12].
Colaborador(es): Instituto de Investigación imas12.
Editor: Biological psychiatry, 2013Descripción: 73(1):32-43.Recursos en línea: Solicitar documento Resumen: BACKGROUND: Stressful challenges are associated with variations in immune parameters, including increased innate immunity/inflammation. Among possible mechanisms through which brain monitors peripheral immune responses, toll-like receptors (TLRs) recently emerged as the first line of defense against invading microorganisms. Their expression is modulated in response to pathogens and other environmental stresses.
| Tipo de ítem | Ubicación actual | Signatura | Estado | Fecha de vencimiento |
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Artículo
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PC12895 (Navegar estantería) | Disponible |
Formato Vancouver:
Gárate I, Garcia-Bueno B, Madrigal JL, Caso JR, Alou L, Gomez-Lus ML et al. Stress-induced neuroinflammation: role of the Toll-like receptor-4 pathway. Biol Psychiatry. 2013 Jan 1;73(1):32-43.
PMID: 22906518
Contiene 65 referencias
BACKGROUND: Stressful challenges are associated with variations in immune parameters, including increased innate immunity/inflammation. Among possible mechanisms through which brain monitors peripheral immune responses, toll-like receptors (TLRs) recently emerged as the first line of defense against invading microorganisms. Their expression is modulated in response to pathogens and other environmental stresses.
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